It has been related with tuberculosis, hypersensitivity to tuberculoprotein, autoimmunity to retinal autoantigens, sure HLA-antigens, oxidative stress of the retina and neurological disorders in different studies; a multifactorial aetiology seems most likely. It is a isobilateral disease, typically affecting young males. The common presenting arguments are sudden appearance of black spots in front of the eye or painless loss of vision due to vitreous haemorrhage. The haemorrhage clears up but frequency very common. There are many treatment of eales disease. Oral corticosteroids are the main remain for treatment during active inflammation.
Laser photocoagulation is argued in the neovascularisation stage of the disease. Vitreoretinal surgery is required for non- concludingvitreous haemorrhage and tractional retinal detachment. Intravitreal triamcinolone acetonide has been efficaciously used in reversing the edema and in leading to visual improvement. Peripheral retinal ischemia and neovascularisations are treated with photocoagulation of the ischemic retina, panretinal photocoagulation or conduct treatment of the neovascularisations. The antioxidant vitamins A, C, and E have been insinuated as a possible therapy because antioxidizing enzymes are deficient in the vitreous samples of patients with Eales disease.
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